Bloodlines
Rh- Blood: Facts & Fiction
http://parablevisions.com/?page_id=525
Byam Shaw (1872-1919) Now is Pilgrim Fair Autumn's Charge
"Splendor Solis" by Gustavo Bueno
IF we assume ---
If Sarah were standing in front of you today, her mtDNA should then show Semitic origins. At least, it would if we assume that her parents Jesus and Mary have the Semitic markers of their community in Israel. One should realize that Dan Brown is not saying that there's anything special about her DNA--just that her very existence would threaten the image of Jesus Christ as established by the Church. So it's not so much the specific CODE of her DNA as the fact that she was even alive--proof that Jesus was a lover and a father. Continuing the hypothesis, let's assume Sarah one day also married and had a child. She would have had to, in fact, for anything in the book to make sense. Well, that child would have nuclear DNA that was 50 percent from Sarah and 50 percent from her father. But her mtDNA would be 100 percent identical to Sarah. So this could be used to track the relationship through time (following the trail of purely female descendents)." --Josh Bernstein
If Sarah were standing in front of you today, her mtDNA should then show Semitic origins. At least, it would if we assume that her parents Jesus and Mary have the Semitic markers of their community in Israel. One should realize that Dan Brown is not saying that there's anything special about her DNA--just that her very existence would threaten the image of Jesus Christ as established by the Church. So it's not so much the specific CODE of her DNA as the fact that she was even alive--proof that Jesus was a lover and a father. Continuing the hypothesis, let's assume Sarah one day also married and had a child. She would have had to, in fact, for anything in the book to make sense. Well, that child would have nuclear DNA that was 50 percent from Sarah and 50 percent from her father. But her mtDNA would be 100 percent identical to Sarah. So this could be used to track the relationship through time (following the trail of purely female descendents)." --Josh Bernstein
Rh- Theories:
http://www.lawpundit.com/blog/2006/08/environment-and-laws-of-genetics-rh.htm
Rh protein plays a significant role as a channel for CO2 gas (carbon dioxide) across cell membranes in the body:
"Rh proteins act as gas channels that help speed the transfer of carbon dioxide (CO2) in and out of red blood cells. CO2 can also pass through the cell membrane unaided (above right), but not quickly enough."
The PubMed Abstract:
"Department of Plant and Microbial Biology, 111 Koshland Hall, University of California, Berkeley, Berkeley, CA 94720-3102, USA.
Physiological evidence from our laboratory indicates that Amt/Mep proteins are gas channels for NH3, the first biological gas channels to be described. This view has now been confirmed by structural evidence and is displacing the previous belief that Amt/Mep proteins were active transporters for the NH4+ ion. Still disputed is the physiological substrate for Rh proteins, the only known homologues of Amt/Mep proteins. Many think they are mammalian ammonium (NH4+ or NH3) transporters. Following Monod's famous dictum, "Anything found to be true of E. coli must also be true of elephants" [Perspect. Biol. Med. 47(1) (2004) 47], we explored the substrate for Rh proteins in the unicellular green alga Chlamydomonas reinhardtii. C. reinhardtii is one of the simplest organisms to have Rh proteins and it also has Amt proteins. Physiological studies in this microbe indicate that the substrate for Rh proteins is CO2 and confirm that the substrate for Amt proteins is NH3. Both are readily hydrated gases. Knowing that transport of CO2 is the ancestral function of Rh proteins supports the inference from hematological research that a newly evolving role of the human Rh30 proteins, RhCcEe and RhD, is to help maintain the flexible, flattened shape of the red cell.
PMID: 16563833 [PubMed - in process]"
Hence, it would seem to be a likely hypothesis to this observer, presented here for the first time, that Rh- (Rh-negative) developed due to a (presumably beneficial) change mandated in our human breathing of the Earth's air in the more northerly European latitudes.
"Temperate and polar oceans of the both hemispheres are the major sinks for atmospheric CO2, whereas the equatorial oceans are the major sources for CO2. The Atlantic Ocean is the most important CO2 sink, providing about 60% of the global ocean uptake, while the Pacific Ocean is neutral because of its equatorial source flux being balanced by the sink flux of the temperate oceans. The Indian and Southern Oceans take up about 20% each."
Given the fact that "the Rh polypeptide is a major fatty acid-acylated erythrocyte membrane protein", i.e. an element of our red blood cells - which transport oxygen to the blood, the discovery that Rh proteins act as gas channels for carbon dioxide in living organisms is one of the most important discoveries made in medicine (and genetics) - ever.
http://www.lawpundit.com/blog/2006/08/environment-and-laws-of-genetics-rh.htm
Rh protein plays a significant role as a channel for CO2 gas (carbon dioxide) across cell membranes in the body:
"Rh proteins act as gas channels that help speed the transfer of carbon dioxide (CO2) in and out of red blood cells. CO2 can also pass through the cell membrane unaided (above right), but not quickly enough."
The PubMed Abstract:
"Department of Plant and Microbial Biology, 111 Koshland Hall, University of California, Berkeley, Berkeley, CA 94720-3102, USA.
Physiological evidence from our laboratory indicates that Amt/Mep proteins are gas channels for NH3, the first biological gas channels to be described. This view has now been confirmed by structural evidence and is displacing the previous belief that Amt/Mep proteins were active transporters for the NH4+ ion. Still disputed is the physiological substrate for Rh proteins, the only known homologues of Amt/Mep proteins. Many think they are mammalian ammonium (NH4+ or NH3) transporters. Following Monod's famous dictum, "Anything found to be true of E. coli must also be true of elephants" [Perspect. Biol. Med. 47(1) (2004) 47], we explored the substrate for Rh proteins in the unicellular green alga Chlamydomonas reinhardtii. C. reinhardtii is one of the simplest organisms to have Rh proteins and it also has Amt proteins. Physiological studies in this microbe indicate that the substrate for Rh proteins is CO2 and confirm that the substrate for Amt proteins is NH3. Both are readily hydrated gases. Knowing that transport of CO2 is the ancestral function of Rh proteins supports the inference from hematological research that a newly evolving role of the human Rh30 proteins, RhCcEe and RhD, is to help maintain the flexible, flattened shape of the red cell.
PMID: 16563833 [PubMed - in process]"
Hence, it would seem to be a likely hypothesis to this observer, presented here for the first time, that Rh- (Rh-negative) developed due to a (presumably beneficial) change mandated in our human breathing of the Earth's air in the more northerly European latitudes.
"Temperate and polar oceans of the both hemispheres are the major sinks for atmospheric CO2, whereas the equatorial oceans are the major sources for CO2. The Atlantic Ocean is the most important CO2 sink, providing about 60% of the global ocean uptake, while the Pacific Ocean is neutral because of its equatorial source flux being balanced by the sink flux of the temperate oceans. The Indian and Southern Oceans take up about 20% each."
Given the fact that "the Rh polypeptide is a major fatty acid-acylated erythrocyte membrane protein", i.e. an element of our red blood cells - which transport oxygen to the blood, the discovery that Rh proteins act as gas channels for carbon dioxide in living organisms is one of the most important discoveries made in medicine (and genetics) - ever.
Thomas Cooper Gotch (British, 1854-1931), "The Child in the World"
In order to express a recessive trait you must have two recessive genes. It could be ten or more generations since anyone has had Rh- blood in your family. But the RhD- version of the RhD gene is still there. http://genetics.thetech.org/ask/ask433
Each person receives an A, B, or O gene from each parent. In this system, the A and B genes are co-dominant and the O gene is recessive. Thus, a person whose genetic type is either AA or AO will have blood type A, those with genetic type BB or BO will have blood type B, and only those genetic type OO will have blood type O. This means that a child with type O blood could have parents with type A, type B, or type O blood (but not with type AB). Conversely, if two parents both have type O blood, all their children will have type O blood. Another medically important blood type is described in the Rh system.
The Rh system is actually far more complex than the ABO system; there are 35 different possibilities that one could inherit from each parent. These, however, are roughly grouped into positive and negative types. In this system the positive are dominant over the negative. If your genetic type is ++ or +-, your blood type will be Rh positive. Only if your genetic type is -- will you be Rh negative. This means that if both parents have Rh+ blood with the +- genes, they could have children who are ++, +-, or --. In other words, their children could be either Rh positive or Rh negative. Children who are Rh negative can have parents who are either Rh positive or Rh negative.
Two parents who have O positive blood could easily have a child who is O negative. In fact, most children who are O negative have parents who are positive, since the +- combination is so much more common than the -- combination.
The father's blood type is important only if a mom is Rh-negative. Her baby will inherit a blood type from her husband. The Rh factor is a dominant gene. If the mother is Rh negative, it means she has no rh factor and can only contribute a gene with an Rhnegative blood type; therefore her husband's genes will be responsible for the blood type of the baby.
If the father is also Rh-negative, then all their babies will also be Rh-negative. There will never be a problem with Rh-disease in their pregnancies. But if the father is Rh-positive then this means he has the Rh factor and can father Rhpositive babies, though he still "might" carry a recessive Rh-negative gene. (There is a blood test to discover this).
If he is Rh-positive but does not carry the recessive gene, then every one of their babies will be Rh-positive and at risk of Rh-disease. But if he carries the Rh-negative recessive gene, then there is a chance that some of their children CAN be Rh-negative.
Q: Neither of my parents have RH Negative blood type but my sister does, how can that be?
Best Answer - Chosen by Voters
Rh- is a recessive gene to Rh+.
If you don't remember anything from high school biology, here's a simple explanation of what that means:
When a child is born, they are given two genes of each trait: One from each parent. The genes can either be dominant, recessive or co-dominant. Dominant means that it hides the traits of the recessive gene. Recessive means that it is masked by the dominant trait but, if it is matched with a recessive gene, the child will have this trait. Co-dominant means that if a child receives two of these, the traits mesh together. (Example: When black and white rabbits breed, they come up with babies that are spotted.)
The Rh- gene is recessive to the Rh+ gene. Your parents must be of the genotype Rh+-. Because each parent is carrying both the positive and the negative gene, but the negative gene is recessive to the positive gene, the parent could pass on either the Rh+ or the Rh- gene.
Your sister received both Rh- genes from both of her parents so that makes her genotype Rh--. This endows her with Rh- blood.
Absolutely yes a RH- child can have two Rh+ parents. This is an example of Mendelian genetics (i.e. dominant vs recessive). The "rhesus factor" is a protein on the surface of red blood cells. Positive means that you make the protein, negative means that you don't. Since you only need one functioning copy of the gene for this protein to make the protein, the functional gene is "dominant." Someone who is rhesus positive can have either 1 or 2 functional copies of the gene. In genetics, this is written "RR" or "Rr," where the capital letter represents the functional copy of the gene. When two people who are "Rr" have children, each child inherits one copy of the gene from each parent. Each parent can pass on either the "R" or the "r" version. The chance that each child will be "rr" and be Rh negative is 25%. The "r" version of this gene is pretty common, especially in Caucasians, so it is not unusual for people who are Rh positive to have one copy of the "r" version of the gene.
Example: I had a family of 4 boys, the first two children were born 0- rh negative and quite well, next birth was twin boys who were born premature 7mths and badly jaundiced, at a later date when I became interested in this blood group thing, I found my first two births were O rh neg, and later my twins boys were O+ rh positive, carrying the rh negative gene...( badly jaundiceD and premature) so if they had gone full term they would not have survived, not much was known in those early days, the Dr just told me not to have any more children? BUT.. nothing about incompatable blood! aha.....re medical research data, my husband was rh positive carrying the recessive rh negative gene, ( his ancestors are from SPAIN) an area of high concentration of rh negative blood types .
....
One of my sons who is an 0 positive blood type but carrying the rh negative recessive gene, married a B rh negative blood lady......he has some O rh negative blood children and some of the children are rh positve.
The World Rh negative blood line can be traced to its 2nd highest concentration, the Basques, on the border of SPAIN/FRANCE..(where our BLOODLINE originates....then to the Middle East..MORROCO > IRAQ the highest concentration of Rh negative blood in the world...adding SUMER into the equation.
Many royal lineage babies who were stillborn or died very shortly after birth, (severely jaundiced), were the end result of mixing of incompatible bloodlines. In previous generations, couples with this problem lost many babies. After it was discovered what was happening (usually the second pregnancy with an Rh pos fetus), couples were advised to not have more biological children; and they followed this advice as the alternative was repeated miscarriages and heartbreak.
What is so important about O Negative bloodlines?Group O, Rh Negative blood has two very distinctive characteristics:
Because of these two characteristics, O Negative blood is mistakenly considered a "universal donor" and can safely be given to ALMOST any recipient. Some people have the belief that O Negative blood is "pure" or "alien". Neither of the latter two statements are true.
Contrary to popular belief, O Negative blood is NOT a universal donor at all and cannot be dispensed to everyone. There are many, many genetic markers other than the "Rh" or "D" antigen marker that must be considered when determining compatibility of blood.
There is a genetic mutation called the "Du" variant. It is actually an Rh Positive antigen, which would make the blood "Rh Positive," but since it is very weakly expressed in tests, it's possible to be mistakenly read as Rh Negative.
Did the Rh factor come from monkeys? The Rh protein and genetic marker was DISCOVERED in the Rhesus monkey. This specific genetic marker is found in primates of which man is one. This indicates that somewhere back in history, the genetic marker came from an ancestor that was common to apes, monkeys and man.
The Rh factor was so named because the discovery was made using Rhesus monkey blood. The nomenclature has been changed. Rh Positives are now noted with the capitol letter "D". There is another set of naming nomenclature that designates the homozygous* (having BOTH dominant expressions for Rh / D) as R1R1.
For further discussion, we can simplify the nomenclature to be:
DD = homozygous (inheriting the Rh factor from both parents)
Dd = heterozygous (inheriting one dominant and one recessive gene for the Rh factor) - Technically, this is actually a hemizygous gene as the lower case "d" marker is actually missing.
dd = the nullizygous expression of the Rh factor. (inheriting only the recessive expressions)
Explanation:
* If both genes (from both parents) are the same, the organism is homozygous for the trait. If both genes are different, the organism is heterozygous for that trait. If one gene is missing, it is hemizygous, and if both genes are missing, it is nullizygous ( Definitions from Wikipedia).
Each person receives an A, B, or O gene from each parent. In this system, the A and B genes are co-dominant and the O gene is recessive. Thus, a person whose genetic type is either AA or AO will have blood type A, those with genetic type BB or BO will have blood type B, and only those genetic type OO will have blood type O. This means that a child with type O blood could have parents with type A, type B, or type O blood (but not with type AB). Conversely, if two parents both have type O blood, all their children will have type O blood. Another medically important blood type is described in the Rh system.
The Rh system is actually far more complex than the ABO system; there are 35 different possibilities that one could inherit from each parent. These, however, are roughly grouped into positive and negative types. In this system the positive are dominant over the negative. If your genetic type is ++ or +-, your blood type will be Rh positive. Only if your genetic type is -- will you be Rh negative. This means that if both parents have Rh+ blood with the +- genes, they could have children who are ++, +-, or --. In other words, their children could be either Rh positive or Rh negative. Children who are Rh negative can have parents who are either Rh positive or Rh negative.
Two parents who have O positive blood could easily have a child who is O negative. In fact, most children who are O negative have parents who are positive, since the +- combination is so much more common than the -- combination.
The father's blood type is important only if a mom is Rh-negative. Her baby will inherit a blood type from her husband. The Rh factor is a dominant gene. If the mother is Rh negative, it means she has no rh factor and can only contribute a gene with an Rhnegative blood type; therefore her husband's genes will be responsible for the blood type of the baby.
If the father is also Rh-negative, then all their babies will also be Rh-negative. There will never be a problem with Rh-disease in their pregnancies. But if the father is Rh-positive then this means he has the Rh factor and can father Rhpositive babies, though he still "might" carry a recessive Rh-negative gene. (There is a blood test to discover this).
If he is Rh-positive but does not carry the recessive gene, then every one of their babies will be Rh-positive and at risk of Rh-disease. But if he carries the Rh-negative recessive gene, then there is a chance that some of their children CAN be Rh-negative.
Q: Neither of my parents have RH Negative blood type but my sister does, how can that be?
Best Answer - Chosen by Voters
Rh- is a recessive gene to Rh+.
If you don't remember anything from high school biology, here's a simple explanation of what that means:
When a child is born, they are given two genes of each trait: One from each parent. The genes can either be dominant, recessive or co-dominant. Dominant means that it hides the traits of the recessive gene. Recessive means that it is masked by the dominant trait but, if it is matched with a recessive gene, the child will have this trait. Co-dominant means that if a child receives two of these, the traits mesh together. (Example: When black and white rabbits breed, they come up with babies that are spotted.)
The Rh- gene is recessive to the Rh+ gene. Your parents must be of the genotype Rh+-. Because each parent is carrying both the positive and the negative gene, but the negative gene is recessive to the positive gene, the parent could pass on either the Rh+ or the Rh- gene.
Your sister received both Rh- genes from both of her parents so that makes her genotype Rh--. This endows her with Rh- blood.
Absolutely yes a RH- child can have two Rh+ parents. This is an example of Mendelian genetics (i.e. dominant vs recessive). The "rhesus factor" is a protein on the surface of red blood cells. Positive means that you make the protein, negative means that you don't. Since you only need one functioning copy of the gene for this protein to make the protein, the functional gene is "dominant." Someone who is rhesus positive can have either 1 or 2 functional copies of the gene. In genetics, this is written "RR" or "Rr," where the capital letter represents the functional copy of the gene. When two people who are "Rr" have children, each child inherits one copy of the gene from each parent. Each parent can pass on either the "R" or the "r" version. The chance that each child will be "rr" and be Rh negative is 25%. The "r" version of this gene is pretty common, especially in Caucasians, so it is not unusual for people who are Rh positive to have one copy of the "r" version of the gene.
Example: I had a family of 4 boys, the first two children were born 0- rh negative and quite well, next birth was twin boys who were born premature 7mths and badly jaundiced, at a later date when I became interested in this blood group thing, I found my first two births were O rh neg, and later my twins boys were O+ rh positive, carrying the rh negative gene...( badly jaundiceD and premature) so if they had gone full term they would not have survived, not much was known in those early days, the Dr just told me not to have any more children? BUT.. nothing about incompatable blood! aha.....re medical research data, my husband was rh positive carrying the recessive rh negative gene, ( his ancestors are from SPAIN) an area of high concentration of rh negative blood types .
....
One of my sons who is an 0 positive blood type but carrying the rh negative recessive gene, married a B rh negative blood lady......he has some O rh negative blood children and some of the children are rh positve.
The World Rh negative blood line can be traced to its 2nd highest concentration, the Basques, on the border of SPAIN/FRANCE..(where our BLOODLINE originates....then to the Middle East..MORROCO > IRAQ the highest concentration of Rh negative blood in the world...adding SUMER into the equation.
Many royal lineage babies who were stillborn or died very shortly after birth, (severely jaundiced), were the end result of mixing of incompatible bloodlines. In previous generations, couples with this problem lost many babies. After it was discovered what was happening (usually the second pregnancy with an Rh pos fetus), couples were advised to not have more biological children; and they followed this advice as the alternative was repeated miscarriages and heartbreak.
What is so important about O Negative bloodlines?Group O, Rh Negative blood has two very distinctive characteristics:
- It is lacking either the "A" antigen or the "B" antigen in the ABO Blood Grouping classification developed by Karl Landsteiner for the major designation of blood groups. There are four major blood groups called Group A, Group B, Group AB and Group O
- It is lacking the "Rh" factor or "D" antigen (also expressed as "R1R1"). This factor was discovered by Landsteiner and others while working with Rhesus monkey blood.
Because of these two characteristics, O Negative blood is mistakenly considered a "universal donor" and can safely be given to ALMOST any recipient. Some people have the belief that O Negative blood is "pure" or "alien". Neither of the latter two statements are true.
Contrary to popular belief, O Negative blood is NOT a universal donor at all and cannot be dispensed to everyone. There are many, many genetic markers other than the "Rh" or "D" antigen marker that must be considered when determining compatibility of blood.
There is a genetic mutation called the "Du" variant. It is actually an Rh Positive antigen, which would make the blood "Rh Positive," but since it is very weakly expressed in tests, it's possible to be mistakenly read as Rh Negative.
Did the Rh factor come from monkeys? The Rh protein and genetic marker was DISCOVERED in the Rhesus monkey. This specific genetic marker is found in primates of which man is one. This indicates that somewhere back in history, the genetic marker came from an ancestor that was common to apes, monkeys and man.
The Rh factor was so named because the discovery was made using Rhesus monkey blood. The nomenclature has been changed. Rh Positives are now noted with the capitol letter "D". There is another set of naming nomenclature that designates the homozygous* (having BOTH dominant expressions for Rh / D) as R1R1.
For further discussion, we can simplify the nomenclature to be:
DD = homozygous (inheriting the Rh factor from both parents)
Dd = heterozygous (inheriting one dominant and one recessive gene for the Rh factor) - Technically, this is actually a hemizygous gene as the lower case "d" marker is actually missing.
dd = the nullizygous expression of the Rh factor. (inheriting only the recessive expressions)
Explanation:
* If both genes (from both parents) are the same, the organism is homozygous for the trait. If both genes are different, the organism is heterozygous for that trait. If one gene is missing, it is hemizygous, and if both genes are missing, it is nullizygous ( Definitions from Wikipedia).
(c)2013; All Rights Reserved, Iona Miller, Sangreality Trust
[email protected]
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[email protected]
Fair Use Notice
This site contains copyrighted material the use of which has not always been specifically authorized by the copyright owner. We are making such material available in our efforts to advance understanding of environmental, political, human rights, economic, democracy, scientific, and social justice issues, etc. We believe this constitutes a 'fair use' of any such copyrighted material as provided for in section 107 of the US Copyright Law. In accordance with Title 17 U.S.C. Section 107, the material on this site is distributed without profit to those who have expressed a prior interest in receiving the included information for research and educational purposes. If you wish to use copyrighted material from this site for purposes of your own that go beyond 'fair use', you must obtain permission from the copyright owner.